Category: Diet

High protein diet and metabolism

High protein diet and metabolism

Supplier IHgh. Heather Lean chicken breast, MS, RDN, Guarana tea benefits. References Halton TL, Hu FB: The effects of High protein diet and metabolism protein diets on thermogenesis, satiety Hiyh weight loss: a critical review. Article CAS Google Scholar Templeman, I. Reprints and permissions. Nutr Metab Lond 1153 Comparison of high protein and high fiber weight-loss diets in women with risk factors for the metabolic syndrome: a randomized trial. High protein diet and metabolism

High protein diet and metabolism -

You burn more calories when eating protein since your body is working harder to digest the food. This is also known as the thermic effect of food. Aside from scientific studies about why protein is so good for your metabolism, it boils down to something simple. What exactly is a high protein diet?

Use one day out of the week to meal prep high protein meals for lunch or dinner. This could mean grilling chicken and roasting vegetables for the week. Just added to your cart. Continue shopping View cart. These Diets Can Help You Lose Weight, For Good. How To Lose Weight After Skip to Content Health Fitness Beauty Life Relationships.

sign in. MORE: 7 High-Protein Snacks That Can Help You Lose Weight It's unclear exactly how protein changes the way the body stores calories, but this study suggests that protein may have a huge impact on your body-fat percentage.

MORE: 4 Myths About Your Metabolism So how much protein should you be eating regularly? Watch Next. How To Lose Weight. From Women's Health for OPTAVIA. Kolodziejczyk, A. Diet-microbiota interactions and personalized nutrition. Schroeder, B. Signals from the gut microbiota to distant organs in physiology and disease.

Das, N. Microbial metabolite signaling is required for systemic iron homeostasis. Quan, L. Myristoleic acid produced by enterococci reduces obesity through brown adipose tissue activation.

Gut 69 , — Araújo, J. Fermentation products of commensal bacteria alter enterocyte lipid metabolism. Cell Host Microbe 27 , — Canfora, E. Gut microbial metabolites in obesity, NAFLD and T2DM.

Choi, H. Gut microbiota as a transducer of dietary cues to regulate host circadian rhythms and metabolism.

Li, G. Intermittent fasting promotes white adipose browning and decreases obesity by shaping the gut microbiota. Thaiss, C. Persistent microbiome alterations modulate the rate of post-dieting weight regain.

de Cabo, R. Effects of intermittent fasting on health, aging, and disease. Bray, M. Quantitative analysis of light-phase restricted feeding reveals metabolic dyssynchrony in mice.

de Aguiar Vallim, T. Pleiotropic roles of bile acids in metabolism. Chiang, J. Bile acids as metabolic regulators and nutrient sensors. Kuang, Z. The intestinal microbiota programs diurnal rhythms in host metabolism through histone deacetylase 3. Fabbiano, S.

Functional gut microbiota remodeling contributes to the caloric restriction-induced metabolic improvements. Zarrinpar, A. Antibiotic-induced microbiome depletion alters metabolic homeostasis by affecting gut signaling and colonic metabolism.

Dodd, D. A gut bacterial pathway metabolizes aromatic amino acids into nine circulating metabolites.

Custers, K. Television viewing, computer game play and book reading during meals are predictors of meal skipping in a cross-sectional sample of , and year-olds. Public Health Nutr. Liechty, J. Longitudinal predictors of dieting and disordered eating among young adults in the U.

Turicchi, J. Associations between the rate, amount, and composition of weight loss as predictors of spontaneous weight regain in adults achieving clinically significant weight loss: a systematic review and meta-regression.

Engber, D. Unexpected clues emerge about why diets fail. Melby, C. Attenuating the biologic drive for weight regain following weight loss: must what goes down always go back up? Nutrients 9 , Fothergill, E. Obesity 24 , — Vink, R. The effect of rate of weight loss on long-term weight regain in adults with overweight and obesity.

Kleinert, M. Animal models of obesity and diabetes mellitus. Ko, C. Regulation of intestinal lipid metabolism: current concepts and relevance to disease.

MacLean, P. The role for adipose tissue in weight regain after weight loss. van Baak, M. Mechanisms of weight regain after weight loss - the role of adipose tissue.

Brehm, B. Benefits of high-protein weight loss diets: enough evidence for practice? Diabetes Obes. Westerterp-Plantenga, M. Dietary protein - its role in satiety, energetics, weight loss and health. Pan, F. Predominant gut Lactobacillus murinus strain mediates anti-inflammaging effects in calorie-restricted mice.

Microbiome 6 , 54 Borthakur, A. María Remes Troche, J. Lactobacillus acidophilus LB: a useful pharmabiotic for the treatment of digestive disorders. Hempel, S. Probiotics for the prevention and treatment of antibiotic-associated diarrhea: a systematic review and meta-analysis.

JAMA , — Talukdar, S. Neutrophils mediate insulin resistance in mice fed a high-fat diet through secreted elastase. Liu, Y. Liver Patt1 deficiency protects male mice from age-associated but not high-fat diet-induced hepatic steatosis. Lipid Res. Khalifeh-Soltani, A. Mfge8 promotes obesity by mediating the uptake of dietary fats and serum fatty acids.

Bolyen, E. Reproducible, interactive, scalable and extensible microbiome data science using QIIME 2. Segata, N. Metagenomic biomarker discovery and explanation.

Genome Biol. Morton, J. Establishing microbial composition measurement standards with reference frames. Wilson, K.

Preparation of genomic DNA from bacteria. mbs56 Nadkarni, M. Determination of bacterial load by real-time PCR using a broad-range universal probe and primers set. Microbiology , — Kuss, S. Intestinal microbiota promote enteric virus replication and systemic pathogenesis.

Chen, C. Complete genome sequence of the probiotic bacterium Lactobacillus casei LC2W. Kuhl, C. Download references. This work was supported by grants from the National Natural Science Foundation of China to Q. XDB , the Frontier Science of Chinese Academy of Sciences Key Research Projects to Q.

QYZDJ-SSW-SMC , the Shanghai Leading Talents Program to Q. and the China Postdoctoral Science Foundation Grant to Y. CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.

Key Laboratory of Synthetic Biology, CAS Center for Excellence in Molecular Plant Sciences, Chinese Academy of Sciences, Shanghai, China. National Clinical Research Center for Eye Diseases, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China. Zhongshan Hospital, State Key Laboratory of Medical Neurobiology, Institute for Translational Brain Research, Ministry of Education Frontiers Center for Brain Science, Fudan University, Shanghai, China.

School of Life Science and Technology, ShanghaiTech University, Shanghai, China. You can also search for this author in PubMed Google Scholar. and Q. participated in data interpretation and revising the paper and approved the final version of the manuscript.

performed most of the experiments. performed absolute quantification of amino acids, fatty acids and metabolites using MS. and Y. measured body composition of mice. and B. helped with experiments using germ-free and gnotobiotic mice. and N. collected tissues of mice.

designed the study, analyzed the data and wrote the manuscript. supervised the project. Correspondence to Qiwei Zhai. are employees of GemPharmatech and T. has stakeholder interests.

All other authors have no competing interests. Nature Metabolism thanks the anonymous reviewers for their contribution to the peer review of this work. Arrows show the time points to provide food, to measure body weight, body composition and food intake.

Each mouse was housed separately for 5 days before the SDR experiment. ZT, Zeitgeber time. ZT0 indicates the beginning of light time, ZT12 indicates the beginning of dark time.

b , c , Refeeding after feeding with 1-day food in 3 days or 4-day food in 6 days markedly increased body fat percentage b and decreased lean mass percentage c. d , e , Body lean mass d and daily food intake e of mice in b. Arrows show the time points to provide food, to collect blood and tissue samples.

Yellow arrow heads indicate the inguinal adipose tissues iWAT. h , i , Representative images of isolated inguinal white adipose tissue iWAT h and epididymal white adipose tissue eWAT i from mice in g.

k , Average adipocyte area of iWAT in Fig. Exact P values can be found in Source Data Extended Data Fig. a , Experimental design of short-term dietary restriction SDR in mice feeding with 2-day food in 3 days or 1-day food in 2 days.

b , c , Refeeding after feeding with 2-day food in 3 days or 1-day food in 2 days markedly increased fat mass percentage b and decreased lean mass percentage c. d - f , Body weight d , body lean mass e and daily food intake f of mice in b.

g , Experimental design of DR in mice feeding with DR h , i , Refeeding after feeding with v - y , Body temperature of mice fed ad libitum or upon DR for the indicated patterns.

The data shown in h - q or x , y were performed simultaneously with a single control experiment. a - d , Cumulative food intake of mice fed ad libitum and upon DR for the indicated patterns.

The average log 2 value of each gene in D0 group was set to 0. Samples in D4 or D6 group were shown in triplicate and normalized to the average expression level in D0 group. The data shown in a , Fig. NP, normal-protein diet; LP, low-protein diet.

e , f , Body weight e and body lean mass f of mice in c. g , h , Representative images of isolated iWAT g and eWAT h from mice as treated with NP or high-protein diet after SDR in c. j , Average adipocyte area of iWAT in Fig. m , Representative images of isolated iBAT from mice in g.

a , b , A high-protein diet maintained the decrease of fat mass percentage a and the increase of lean mass percentage b induced by feeding with e , f , A high-protein diet maintained the decrease of fat mass percentage e and the increase of lean mass percentage f induced by feeding with g , h , Body weight g and body lean mass h of mice in e.

m , n , Cumulative food intake of male mice fed ad libitum AL or fed with NP or high-protein diet after dieting with q - s , Daily food intake of mice in m-o.

a , Composition of normal-protein NP , high-protein HP and high-fat HF diets represented in percent calories. b , Food intake of mice fed ad libitum, mice refeeding with NP or HP diet after SDR and mice pair-fed with the isocaloric NP or HF diet after SDR.

c , d , Pair-feeding with normal-protein or high-fat diet after SDR markedly increased body fat percentage c and decreased lean mass percentage d.

e , f , Body weight e and body lean mass f of mice in b. The average log 2 value of each gene in D0 NP group was set to 0. a , Experimental design of mice gavaged with or without a mixture of antibiotics ABX.

Arrows show the time points of water or ABX gavage, feces collection, BODIPY-labeled fatty acid gavage and sampling. SPF, specific pathogen-free.

g , Representative fluorescent section images of iWAT and eWAT from mice in d. NS, not significant. a , Experimental design of fecal microbiota transplantation FMT from the donor mice into recipient mice. Fecal microbiota FM from mice in AL-NP, SDR-NP or SDR-HP group were transplanted into SPF recipient mice.

f , g , Small intestine f and serum g BODIPY concentrations of mice in e. h , Representative fluorescent section images of iWAT and eWAT from mice in e. Springer Nature or its licensor e. a society or other partner holds exclusive rights to this article under a publishing agreement with the author s or other rightsholder s ; author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law.

Reprints and permissions. Zhong, W. High-protein diet prevents fat mass increase after dieting by counteracting Lactobacillus -enhanced lipid absorption. Nat Metab 4 , — Download citation. Received : 12 October Accepted : 14 October Published : 01 December Issue Date : December Anyone you share the following link with will be able to read this content:.

Sorry, a shareable link is not currently available for this article. Provided by the Springer Nature SharedIt content-sharing initiative. Sign up for the Nature Briefing newsletter — what matters in science, free to your inbox daily.

Skip to main content Thank you for visiting nature. nature nature metabolism articles article. Download PDF. Subjects Fat metabolism Metabolism Obesity.

Abstract Dietary restriction is widely used to reduce fat mass and lose weight in individuals with or without obesity; however, weight regain after dieting is still a big challenge, and the underlying mechanisms remain largely elusive. Hydrogen-rich water reduces inflammatory responses and prevents apoptosis of peripheral blood cells in healthy adults: a randomized, double-blind, controlled trial Article Open access 22 July Microbiota in health and diseases Article Open access 23 April Effects of dietary fibre on metabolic health and obesity Article 07 February Main The global prevalence of obesity has tripled since the mids 1.

Full size image. Discussion Here we show that refeeding after dieting promotes the enrichment of intestinal Lactobacillus and its metabolites, which can be blocked by HP diet or antibiotics, thereby enhances intestinal lipid absorption and fatty acid uptake in WAT and leads to increased fat mass gain Fig.

Methods Dietary restriction experiments All animals were maintained and used in accordance with the guidelines of and under approval by, the Institutional Animal Care and Use Committee of Shanghai Institute of Nutrition and Health, Chinese Academy of Sciences ethical committee approval nos.

Body weight and body composition measurement Body weight and body composition of mice were measured at ZT Body temperature measurement Body temperature of mice was measured at ZT3 using a RET-3 rectal probe Physitemp attached to a BAT microprobe thermometer Physitemp.

Metabolic rate and physical activity Metabolic rate and physical activity of the indicated mice were measured using a chamber environment-controlled Comprehensive Lab Animal Monitoring System with Oxymax software v.

Quantitative PCR Total RNA of hypothalamus was extracted with TRIzol Thermo Fisher, and cDNA was synthesized with M-MLV Reverse Transcriptase Promega, M Measurement of triglyceride Hepatic, small intestinal and fecal TG was extracted and detected as previously described with minor modifications High-throughput RNA sequencing and analysis The indicated tissues were collected at ZT10 to extract total RNA with TRIzol Reagent.

Serum amino acid measurement Serum amino acids were treated with sulfosalicylic acid and labeled with aTRAQ reagent using an aTRAQ Reagent kit AB Sciex. Bile acid analysis Blood and whole intestines, including their contents with the exception of cecal feces, were collected at ZT Isolation of L.

Bacterial DNA extraction and quantification Cecal contents of mice refeeding with NP or HP diet after SDR or feces of mice upon SDR with or without antibiotic treatments were collected at the indicated time points.

Antibiotic susceptibility testing Puromycin Yeasen Biotech, ES25 , vancomycin Meilunbio, MB , neomycin A , metronidazole A , kanamycin A , chloramphenicol A , tetracycline A , ampicillin A and penicillin A from Sangon Biotech were used to detect their effects on the growth of Lactobacillus Lam Antibiotic treatments Antibiotic mixture treatments were performed as described previously with modifications Non-targeted metabolomics and data analysis Blood and cecal feces were collected at ZT Targeted quantitative analysis of PLA, HPLA, ILA, HICA and HMBA E.

Statistical analysis Data are expressed as mean ± s. Reporting summary Further information on research design is available in the Nature Portfolio Reporting Summary linked to this article. References Kumanyika, S.

Article Google Scholar Blüher, M. Article Google Scholar Gao, M. Article CAS Google Scholar Heymsfield, S. Article CAS Google Scholar van der Klaauw, A. Article Google Scholar Pillon, N. Article CAS Google Scholar Franks, P. Article CAS Google Scholar McHill, A.

Article Google Scholar Milaneschi, Y. Article CAS Google Scholar Chao, A. Article CAS Google Scholar Di Francesco, A. Article Google Scholar Lettieri-Barbato, D.

Article CAS Google Scholar Most, J. Article Google Scholar Speakman, J. Article Google Scholar Santos, I. Article CAS Google Scholar Templeman, I. Article Google Scholar Kraus, W. Article Google Scholar Robertson, L. Article CAS Google Scholar Nas, A. CAS Google Scholar Pendergast, F.

Article Google Scholar Johnstone, A. Article CAS Google Scholar Maughan, R. Article CAS Google Scholar Trepanowski, J. Article Google Scholar Dulloo, A. Article Google Scholar Lowe, M. Article Google Scholar Montani, J.

Article Google Scholar Zmora, N. Article CAS Google Scholar Albenberg, L. Article CAS Google Scholar Gentile, C. Article CAS Google Scholar Rinninella, E. Article CAS Google Scholar von Schwartzenberg, R. Article Google Scholar Fraumene, C.

Article Google Scholar Chadaideh, K.

Barbie Cervoni MS, RD, Metaboolism, CDN, is a registered dietitian and aand diabetes High protein diet and metabolism and Wellness supplements specialist. At Verywell, we believe prptein is no one-size-fits-all approach to a healthy lifestyle. Successful eating plans need to be individualized and take the whole person into consideration. Prior to starting a new diet plan, consult with your healthcare provider or a registered dietitian, especially if you have an underlying health condition. Protein is an essential nutrient for health.

High protein diet and metabolism -

Gabby Landsverk. Share icon An curved arrow pointing right. Share Facebook Icon The letter F. Facebook Email icon An envelope. It indicates the ability to send an email. Email Twitter icon A stylized bird with an open mouth, tweeting.

Twitter LinkedIn icon The word "in". LinkedIn Link icon An image of a chain link. It symobilizes a website link url. Copy Link. Redeem now. Eating high-protein foods could help dieters burn calories and fat more efficiently, according to a new study.

Researchers found that a high-protein diet slightly boosted the metabolism of young, healthy volunteers, causing them to burn more calories and fat. That could be because protein takes more energy to digest than carbs or fat, making it a promising tool for weight loss.

More research is needed to see how this works in the long term and for people with obesity. Visit Insider's homepage for more stories. Read preview. Thanks for signing up!

Access your favorite topics in a personalized feed while you're on the go. download the app. Email address. Sign up. You can opt-out at any time. But—get ready for a huge silver lining—those who noshed on normal- and high- protein diets 15 and 25 percent, respectively stored 45 percent of the excess calories as muscle, while those on the low-protein diet five percent stored 95 percent of the excess calories as fat.

MORE: 7 High-Protein Snacks That Can Help You Lose Weight. It's unclear exactly how protein changes the way the body stores calories, but this study suggests that protein may have a huge impact on your body-fat percentage. And since increasing muscle mass can boost metabolism, it stands to reason that eating more protein may help rev yours up.

MORE: 4 Myths About Your Metabolism. So how much protein should you be eating regularly? The study results suggest that for a supercharged metabolism, between 25 percent and 45 percent of your calories should come from protein.

Each gram of protein contains four calories. If you're on a 2,calorie diet, that comes out to about and grams of protein a day, which is similar to the Academy of Nutrition and Dietetics' recommendation to consume 20 to 30 grams of high-quality protein after exercising and every four hours while you're awake to increase your muscle growth.

MORE: 5 Foods with More Protein Than an Egg. Can The Alpilean Ice Diet Help You Lose Weight? Can Magnesium Help You Lose Weight? Do Apple Cider Vinegar Gummies Aid Weight Loss? What Is The Optavia Diet?

Hlgh Clinic Guarana tea benefits appointments in Arizona, Florida and Trim visceral fat and at Mayo Clinic Health System locations. Such diets emtabolism help with weight loss by making you feel fuller. But many health issues may happen if you follow a high-protein diet for a long time. And researchers are still studying the long-term risks of high-protein diets that limit carbohydrates carbs. If you want to stick to a high-protein diet, choose your protein with care.


The Most EFFICIENT Way To LOSE FAT - Andrew Huberman Metrics Dket. High protein mehabolism are increasingly popularized in trim visceral fat media as a High protein diet and metabolism strategy for weight loss by providing the twin benefits of improving satiety Black pepper extract for enhancing nutrient absorption decreasing fat mass. Some of the potential mechanisms that anr for weight loss associated with high-protein diets involve increased secretion of satiety hormones GIP, GLP-1reduced orexigenic hormone secretion ghrelinthe increased thermic effect of food and protein-induced alterations in gluconeogenesis to improve glucose homeostasis. There are, however, also possible caveats that have to be considered when choosing to consume a high-protein diet. A high intake of branched-chain amino acids in combination with a western diet might exacerbate the development of metabolic disease.

Author: Akinorisar

5 thoughts on “High protein diet and metabolism

  1. Nach meiner Meinung lassen Sie den Fehler zu. Ich biete es an, zu besprechen. Schreiben Sie mir in PM, wir werden reden.

Leave a comment

Yours email will be published. Important fields a marked *

Design by